The link between different infection forms of Porphyromonas gingivalis and acute myocardial infarction: a cross-sectional study

Table 4 Correlation between Pg infection and risk of AMI occurrence

Pg (+)	Case number (N)	AMI (N)	Model 1^a		Model 2^b		Model 3^c
Pg (+)	Case number (N)	AMI (N)	OR (95% CI)	p value	OR (95% CI)	p value	OR (95% CI)	p value
Oral cavity	243	209	1.563 (0.957–2.554)	0.74	1.513 (0.900–2.543)	0.12	1.457 (0.853–2.486)	0.17
cfDNA	203	177	1.727 (1.035–2.881)	< 0.05	1.711 (1.005–2.911)	< 0.05	1.671 (0.967–2.890)	0.07
gDNA	172	152	1.917 (1.108–3.315)	< 0.05	1.863 (1.064–3.262)	< 0.05	1.853 (1.042–3.295)	< 0.05

Pg Porphyromonas gingivalis, cfDNA circulating free DNA, gDNA genomic DNA
^aVariables without adjustment
^bAdjusting gender, age, and medical history (hypertension, diabetes, and current smoking)
^cAdjusting the variables in the model^b as well as the low-density lipoprotein, high-density lipoprotein cholesterol, triglyceride, lipoprotein a, and homocysteine

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