Fig. 3From: Depletion of EREG enhances the osteo/dentinogenic differentiation ability of dental pulp stem cells via the p38 MAPK and Erk pathways in an inflammatory microenvironmentKnockdown of EREG activated the p38 MAPK and Erk signalling pathways in DPSCs. a, b Western blot results and quantitative analysis of the expression of phosphorylated p38 MAPK and phosphorylated Erk in EREGsh and Scramsh DPSCs. β-Actin was used as an internal control. c, d Western blot results and quantitative analysis of the expression of phosphorylated Erk in DPSCs after treatment with 20 μmol/L Erk‐specific inhibitor PD98059 for 1 h in DPSCs. β-Actin was used as an internal control. e, f Western blot results and quantitative analysis of the expression of phosphorylated p38 MAPK in DPSCs after treatment with 20 μmol/L p38 MAPK‐specific inhibitor SB203580 for 1 h in DPSCs. β-Actin was used as an internal control. Error bars represent the SD (n = 3). One-way ANOVA with the post hoc Bonferroni test were used to test statistical significance. *P ≤ 0.05. **P ≤ 0.01Back to article page